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Voltage-gated Nachannel ( I) function is critical for normal cardiac excitability. However, the Na channel "late" component ( I) is directly associated with potentially fatal forms of congenital and acquired human arrhythmia. Ca/calmodulin-dependent kinase II (CaMKII) enhances I in response to increased adrenergic tone. However, the pathways that negatively regulate the CaMKII/Nav1.5 axis are unknown and essential for the design of new therapies to regulate the pathogenic I.
PMID: 30602331 [PubMed - as supplied by publisher]
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