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Role of A20/TNFAIP3 deficiency in lupus nephritis in MRL/lpr mice.

著者 Sun L , Zou LX , Han YC , Zhu DD , Chen T , Wang J
Clin Exp Nephrol.2019 Dec 07 ; ():.
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The activation of the nuclear factor-κB (NF-κB) signaling pathway gives rise to inflammation in the pathogenesis of lupus nephritis (LN), with A20 serving as a negative feedback regulator and ubiquitin C‑terminal hydrolase L1 (UCH-L1) acting as a downstream target protein. However, their roles in the mechanism of LN remain undetermined.
PMID: 31811410 [PubMed - as supplied by publisher]
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