絞り込み

16733

広告

Brain imaging measurements of fibrillar amyloid-β burden, paired helical filament tau burden, and atrophy in cognitively unimpaired persons with two, one, and no copies of the APOE ε4 allele.

著者 Ghisays V , Goradia DD , Protas H , Bauer RJ , Devadas V , Tariot PN , Lowe VJ , Knopman DS , Petersen RC , Jack CR , Caselli RJ , Su Y , Chen K , Reiman EM
Alzheimers Dement.2019 Dec 09 ; ():.
この記事をPubMed上で見るPubMedで表示
この記事をGoogle翻訳上で見る Google翻訳で開く

スターを付ける スターを付ける     (1view , 0users)

Full Text Sources

Miscellaneous

We previously characterized associations between brain imaging measurements of amyloid-β (Aβ) plaque burden and apolipoprotein E (APOE) ϵ4 gene dose in a small number of cognitively unimpaired late-middle-aged APOE ϵ4 homozygotes (HMs), heterozygotes (HTs), and noncarriers (NCs). We now characterize cross-sectional Aβ plaque, tau tangle, and cortical atrophy (neurodegeneration) measurements, classifications, and associations with age in a larger number of unimpaired HMs, HTs, and NCs over a wider age range.
PMID: 31831374 [PubMed - as supplied by publisher]
印刷用ページを開く Endnote用テキストダウンロード