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「"beeg m"[Author]」の検索結果

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Utilization of the Monte Carlo method to build up QSAR models for hemolysis and cytotoxicity of antimicrobial peptides.

QSAR model for blood-brain barrier permeation.

Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.

Fingolimod Limits Acute Aβ Neurotoxicity and Promotes Synaptic Versus Extrasynaptic NMDA Receptor Functionality in Hippocampal Neurons.

Cardiac light chain amyloidosis: The role of metal ions in oxidative stress and mitochondrial damage.

The Anti-Prion Antibody 15B3 Detects Toxic Amyloid-β Oligomers.

Monte Carlo method for Predicting of Cardiac Toxicity: hERG blocker compounds.

Clusterin binds to Aβ1-42 oligomers with high affinity and interferes with peptide aggregation by inhibiting primary and secondary nucleation.

The new β amyloid-derived peptide Aβ1-6A2V-TAT(D) prevents Aβ oligomer formation and protects transgenic C. elegans from Aβ toxicity.

Different mutations at V363 MAPT codon are associated with atypical clinical phenotypes and show unusual structural and functional features.

Novel approaches for studying amyloidogenic peptides/proteins.

Specific recognition of biologically active amyloid-β oligomers by a new Surface Plasmon Resonance-based immunoassay and an in vivo assay in Caenorhabditis elegans.

In Vitro Aggregation Behavior of a Non-Amyloidogenic λ Light Chain Dimer Deriving from U266 Multiple Myeloma Cells.

New mutations in MAPT gene causing frontotemporal lobar degeneration: biochemical and structural characterization.

A modified protocol to prepare seed-free starting solutions of amyloid-β (Aβ)₁₋₄₀ and Aβ₁₋₄₂ from the corresponding depsipeptides.

Use of surface plasmon resonance to study the elongation kinetics and the binding properties of the highly amyloidogenic Aβ(1-42) peptide, synthesized by depsi-peptide technique.

Lipid-based nanoparticles with high binding affinity for amyloid-beta1-42 peptide.

Development of a proteolytically stable retro-inverso peptide inhibitor of beta-amyloid oligomerization as a potential novel treatment for Alzheimer's disease.

Synthetic amyloid-{beta} oligomers impair long-term memory independently of cellular prion protein.

A recessive mutation in the APP gene with dominant-negative effect on amyloidogenesis.

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