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「"Chiasson VL "[Author]」の検索結果

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Both maternal and placental toll-like receptor activation are necessary for the full development of proteinuric hypertension in mice.

Myeloid-Derived Suppressor Cells Ameliorate Cyclosporine A-Induced Hypertension in Mice.

MicroRNAs: New Players in the Pathobiology of Preeclampsia.

Depletion of MHC class II invariant chain peptide or γ-δ T-cells ameliorates experimental preeclampsia.

Regulatory T-Cell Augmentation or Interleukin-17 Inhibition Prevents Calcineurin Inhibitor-Induced Hypertension in Mice.

Human placenta-derived stromal cells decrease inflammation, placental injury, and blood pressure in hypertensive pregnant mice.

Cotreatment With Interleukin 4 and Interleukin 10 Modulates Immune Cells and Prevents Hypertension in Pregnant Mice.

Regulation of the Anti-Inflammatory Cytokines Interleukin-4 and Interleukin-10 during Pregnancy.

TLR3-induced placental miR-210 down-regulates the STAT6/interleukin-4 pathway.

Interleukin-4 deficiency induces mild preeclampsia in mice.

Interleukin-17 causes Rho-kinase-mediated endothelial dysfunction and hypertension.

Placental Toll-like receptor 3 and Toll-like receptor 7/8 activation contributes to preeclampsia in humans and mice.

Endothelial cell transforming growth factor-β receptor activation causes tacrolimus-induced renal arteriolar hyalinosis.

Pin1 Deficiency Causes Endothelial Dysfunction and Hypertension.

Interleukin 10 Deficiency Exacerbates Toll-Like Receptor 3-Induced Preeclampsia-Like Symptoms in Mice.

FK506 binding protein 12 deficiency in endothelial and hematopoietic cells decreases regulatory T cells and causes hypertension.

Protein kinase CbetaII-mediated phosphorylation of endothelial nitric oxide synthase threonine 495 mediates the endothelial dysfunction induced by FK506 (tacrolimus).

Do double-stranded RNA receptors play a role in preeclampsia?

Interleukin-10 reduces inflammation, endothelial dysfunction, and blood pressure in hypertensive pregnant rats.

Toll-like receptor 3 activation during pregnancy elicits preeclampsia-like symptoms in rats.

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