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「"Demorrow S "[Author]」の検索結果

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Endothelin inhibits cholangiocarcinoma growth by a decrease in the vascular endothelial growth factor expression.

Taurocholate feeding to bile duct ligated rats prevents caffeic acid-induced bile duct damage by changes in cholangiocyte VEGF expression.

Morphological and functional heterogeneity of the mouse intrahepatic biliary epithelium.

Serotonin metabolism is dysregulated in cholangiocarcinoma, which has implications for tumor growth.

The endocannabinoid anandamide inhibits cholangiocarcinoma growth via activation of the noncanonical Wnt signaling pathway.

Leptin enhances cholangiocarcinoma cell growth.

Progesterone stimulates the proliferation of female and male cholangiocytes via autocrine/paracrine mechanisms.

Small mouse cholangiocytes proliferate in response to H1 histamine receptor stimulation by activation of the IP3/CaMK I/CREB pathway.

Genetic and epigenetic changes associated with cholangiocarcinoma: from DNA methylation to microRNAs.

Anandamide inhibits cholangiocyte hyperplastic proliferation via activation of thioredoxin 1/redox factor 1 and AP-1 activation.

Cholangiocyte injury and ductopenic syndromes.

Biogenic amine actions on cholangiocyte function.

Prolactin stimulates the proliferation of normal female cholangiocytes by differential regulation of Ca2+-dependent PKC isoforms.

The alpha2-adrenergic receptor agonist UK 14,304 inhibits secretin-stimulated ductal secretion by downregulation of the cAMP system in bile duct-ligated rats.

Knockout of alpha-calcitonin gene-related peptide reduces cholangiocyte proliferation in bile duct ligated mice.

Taurocholic acid feeding prevents tumor necrosis factor-alpha-induced damage of cholangiocytes by a PI3K-mediated pathway.

H3 histamine receptor agonist inhibits biliary growth of BDL rats by downregulation of the cAMP-dependent PKA/ERK1/2/ELK-1 pathway.

Opposing actions of endocannabinoids on cholangiocarcinoma growth: recruitment of Fas and Fas ligand to lipid rafts.

Thyroid hormone inhibits biliary growth in bile duct-ligated rats by PLC/IP(3)/Ca(2+)-dependent downregulation of SRC/ERK1/2.

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