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新型コロナで公演中止 俳優や声優は「生きる危機に」 (NHK)

新型コロナウイルスの感染拡大で、演劇などの公演の中止が相次ぐ中、生活のために貯蓄を取り崩したり、借金をしたりする必要があるという俳優や声優が、8割近くにのぼって...

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  3. 「限りある医療資源の選択的配分」について...
  4. COVID-19流行中の癌治療をどうする...

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「"Dickson DW "[Author]」の検索結果

1030件中 121件~140件表示    検索結果をPubMedで見る PubMedで見る

The limbic and neocortical contribution of α-synuclein, tau, and β-amyloid to disease duration in dementia with Lewy bodies.

Establishing diagnostic criteria for Perry syndrome.

Cognitive impairment in progressive supranuclear palsy is associated with tau burden.

Regional analysis and genetic association of nigrostriatal degeneration in Lewy body disease.

Rates of hippocampal atrophy and presence of post-mortem TDP-43 in patients with Alzheimer's disease: a longitudinal retrospective study.

Parkinson's disease susceptibility variants and severity of Lewy body pathology.

DCTN1 variation in pathologically-confirmed PSP and CBD tauopathy.

Recent advances in neuropathology, biomarkers and therapeutic approach of multiple system atrophy.

TIA1 Mutations in Amyotrophic Lateral Sclerosis and Frontotemporal Dementia Promote Phase Separation and Alter Stress Granule Dynamics.

Conserved DNA methylation combined with differential frontal cortex and cerebellar expression distinguishes C9orf72-associated and sporadic ALS, and implicates SERPINA1 in disease.

Brain calcifications and PCDH12 variants.

Atypical Parkinsonian syndromes: a general neurologist's perspective.

Impaired endo-lysosomal membrane integrity accelerates the seeding progression of α-synuclein aggregates.

Perry Syndrome: A Distinctive Type of TDP-43 Proteinopathy.

Neuropathology of Parkinson disease.

An acetylation-phosphorylation switch that regulates tau aggregation propensity and function.

The lysosomal protein cathepsin L is a progranulin protease.

Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease.

Methyl Bromide Alternatives for Control of Root-knot Nematode (Meloidogyne spp.) in Tomato Production in Florida.

Loss of clusterin shifts amyloid deposition to the cerebrovasculature via disruption of perivascular drainage pathways.

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