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「"Diomede L"[Author]」の検索結果

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An across-species comparison of the sensitivity of different organisms to Pb-based perovskites used in solar cells.

Modulating the cardiotoxic behaviour of immunoglobulin light chain dimers through point mutations.

An alternative non-proteolytic mechanism may underlie AGel amyloidosis.

Repeated administration of the food additive E171 to mice results in accumulation in intestine and liver and promotes an inflammatory status.

Nanobody interaction unveils structure, dynamics and proteotoxicity of the Finnish-type amyloidogenic gelsolin variant.

Methacycline displays a strong efficacy in reducing toxicity in a SCA3 Caenorhabditis elegans model.

Biological monitoring of exposure to polycyclic aromatic hydrocarbons and to metallic elements in Italian Navy workers operating near the industrial area in Taranto (South Italy).

Identification of amino acid residues critical for the B cell growth-promoting activity of HIV-1 matrix protein p17 variants.

V363I and V363A mutated tau affect aggregation and neuronal dysfunction differently in C. elegans.

Safety and Toxicology of Magnolol and Honokiol.

Realistic Evaluation of Titanium Dioxide Nanoparticle Exposure in Chewing Gums.

Gelsolin pathogenic Gly167Arg mutation promotes domain-swap dimerization of the protein.

Biological monitoring of exposure to low concentrations of benzene in workers at a metallurgical coke production plant: new insights into S-phenylmercapturic acid and urinary benzene.

HIV-1 matrix protein p17 misfolding forms toxic amyloidogenic assemblies that induce neurocognitive disorders.

Inhibition of Aβ amyloid growth and toxicity by Silybins: the crucial role of stereochemistry.

A simple headspace GC/MS method for the quantitative determination of the release of the antioxidants butylated hydroxyanisole and butylated hydroxytoluene from chewing gum.

Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.

Cardiac light chain amyloidosis: The role of metal ions in oxidative stress and mitochondrial damage.

Murine Rankl(-/-) Mesenchymal Stromal Cells Display an Osteogenic Differentiation Defect Improved by a RANKL-expressing Lentiviral Vector.

SPIRE, a modular pipeline for eQTL analysis of RNA-Seq data, reveals a regulatory hotspot controlling miRNA expression in C. elegans.

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