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「"Kurreeman FA "[Author]」の検索結果

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Mast cells in early rheumatoid arthritis associate with disease severity and support B cell autoantibody production.

The production and secretion of complement component C1q by human mast cells.

Identification of a novel non-coding mutation in C1qB in a Dutch child with C1q deficiency associated with recurrent infections.

Identification of a genetic variant for joint damage progression in autoantibody-positive rheumatoid arthritis.

Genetic variants in the region of the C1q genes are associated with Rheumatoid Arthritis.

Meta-analysis identifies nine new loci associated with rheumatoid arthritis in the Japanese population.

Bayesian inference analyses of the polygenic architecture of rheumatoid arthritis.

Use of a Multiethnic Approach to Identify Rheumatoid- Arthritis-Susceptibility Loci, 1p36 and 17q12.

The major risk alleles of age-related macular degeneration (AMD) in CFH do not play a major role in rheumatoid arthritis (RA).

Fine mapping the TAGAP risk locus in rheumatoid arthritis.

Meta-Analysis of Genome-Wide Association Studies in Celiac Disease and Rheumatoid Arthritis Identifies Fourteen Non-HLA Shared Loci.

Genome-wide association study meta-analysis identifies seven new rheumatoid arthritis risk loci.

TRAF1/C5 polymorphism is not associated with increased mortality in rheumatoid arthritis: two large longitudinal studies.

Evidence for interaction between 5-hydroxytryptamine (serotonin) receptor 2A and MHC type II molecules in the development of rheumatoid arthritis.

Genetic variants at CD28, PRDM1 and CD2/CD58 are associated with rheumatoid arthritis risk.

Association of IL2RA and IL2RB with rheumatoid arthritis: a replication study in a Dutch population.

Association of a single-nucleotide polymorphism in CD40 with the rate of joint destruction in rheumatoid arthritis.

IL-10 promotor haplotypes associated with susceptibility to and severity of bacterial corneal ulcers.

The TRAF1-C5 region on chromosome 9q33 is associated with multiple autoimmune diseases.

Confirmation of STAT4, IL2/IL21, and CTLA4 polymorphisms in rheumatoid arthritis.

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