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「"Lubbers ER "[Author]」の検索結果

23件中 1件~20件表示    検索結果をPubMedで見る PubMedで見る

Aberrant Expression of a Non-muscle RBFOX2 Isoform Triggers Cardiac Conduction Defects in Myotonic Dystrophy.

Correction: Defining new mechanistic roles for α II spectrin in cardiac function.

Ankyrin-B dysfunction predisposes to arrhythmogenic cardiomyopathy and is amenable to therapy.

Defining new mechanistic roles for αII spectrin in cardiac function.

Protein Phosphatase 2A Regulates Cardiac Na Channels.

Novel Mechanistic Roles for Ankyrin-G in Cardiac Remodeling and Heart Failure.

Arrhythmogenic Substrates for Atrial Fibrillation in Obesity.

Advancements in the Use of Gene Therapy for Cardiac Arrhythmia.

Roles and regulation of protein phosphatase 2A (PP2A) in the heart.

Common human ANK2 variant confers in vivo arrhythmia phenotypes.

JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age.

Growth Hormone Receptor Antagonist Transgenic Mice Have Increased Subcutaneous Adipose Tissue Mass, Altered Glucose Homeostasis and No Change in White Adipose Tissue Cellular Senescence.

Defining the links between oxidative stress-based biomarkers and postoperative atrial fibrillation.

GH action influences adipogenesis of mouse adipose tissue-derived mesenchymal stem cells.

Male bovine GH transgenic mice have decreased adiposity with an adipose depot-specific increase in immune cell populations.

Growth hormone receptor antagonist transgenic mice are protected from hyperinsulinemia and glucose intolerance despite obesity when placed on a HF diet.

Growth hormone action predicts age-related white adipose tissue dysfunction and senescent cell burden in mice.

Age-related and depot-specific changes in white adipose tissue of growth hormone receptor-null mice.

A dwarf mouse model with decreased GH/IGF-1 activity that does not experience life-span extension: potential impact of increased adiposity, leptin, and insulin with advancing age.

The Role of GH in Adipose Tissue: Lessons from Adipose-Specific GH Receptor Gene-Disrupted Mice.

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