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「"Mancino MG "[Author]」の検索結果

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Interobserver agreement in contrast harmonic endoscopic ultrasound.

Techniques of image enhancement in EUS (with videos).

Contrast harmonic echo-endoscopic ultrasound improves accuracy in diagnosis of solid pancreatic masses.

Estrogens stimulate the proliferation of human cholangiocarcinoma by inducing the expression and secretion of vascular endothelial growth factor.

New insights on the molecular and cell biology of human cholangiopathies.

Morphological and functional features of hepatic cyst epithelium in autosomal dominant polycystic kidney disease.

[Pathophysiology of cholangiocytes: recent advances].

Serum and biliary insulin-like growth factor I and vascular endothelial growth factor in determining the cause of obstructive cholestasis.

Hepatic "stem" cells: state of the art.

The alpha2-adrenergic receptor agonist UK 14,304 inhibits secretin-stimulated ductal secretion by downregulation of the cAMP system in bile duct-ligated rats.

Bile salts regulate proliferation and apoptosis of liver cells by modulating the IGF1 system.

Proliferating cholangiocytes: a neuroendocrine compartment in the diseased liver.

Activation of the IGF1 system characterizes cholangiocyte survival during progression of primary biliary cirrhosis.

Estrogens and the pathophysiology of the biliary tree.

Ca2+-dependent cytoprotective effects of ursodeoxycholic and tauroursodeoxycholic acid on the biliary epithelium in a rat model of cholestasis and loss of bile ducts.

Adrenergic receptor agonists prevent bile duct injury induced by adrenergic denervation by increased cAMP levels and activation of Akt.

Estrogen receptors in cholangiocytes and the progression of primary biliary cirrhosis.

Nerve growth factor modulates the proliferative capacity of the intrahepatic biliary epithelium in experimental cholestasis.

Gastrin reverses established cholangiocyte proliferation and enhanced secretin-stimulated ductal secretion of BDL rats by activation of apoptosis through increased expression of Ca2+- dependent PKC isoforms.

Dopaminergic inhibition of secretin-stimulated choleresis by increased PKC-gamma expression and decrease of PKA activity.

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