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「"Marzioni M "[Author]」の検索結果

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Aging and the Biological Response to Liver Injury.

Locally acquired hepatitis E virus in Marche Italy: Clinical/laboratory features and outcome.

Secretin/secretin receptor signaling mediates biliary damage and liver fibrosis in early-stage primary biliary cholangitis.

Effects of Vedolizumab in Patients with Primary Sclerosing Cholangitis and Inflammatory Bowel Diseases.

Signaling networks in cholangiocarcinoma: molecular pathogenesis, targeted therapies and drug resistance.

Epidemiology of primary biliary cholangitis in Italy: Evidence from a real-world database.

Ageing-related expression of Twinfilin-1 regulates cholangiocyte biological response to injury.

Wnt-β-catenin signalling in liver development, health and disease.

Inflammation and the Gut-Liver Axis in the Pathophysiology of Cholangiopathies.

Ductular reaction in liver diseases: pathological mechanisms and translational significances.

Pretreatment prediction of response to ursodeoxycholic acid in primary biliary cholangitis: development and validation of the UDCA Response Score.

Knockout of secretin receptor reduces biliary damage and liver fibrosis in Mdr2 mice by diminishing senescence of cholangiocytes.

Safety and efficacy of ombitasvir/paritaprevir/ritonavir/dasabuvir plus ribavirin in patients over 65 years with HCV genotype 1 cirrhosis.

Cholangiocytes in health and disease: From basic science to novel treatments.

Author Correction: Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLD.

Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLD.

MiRNA-506 promotes primary biliary cholangitis-like features in cholangiocytes and immune activation.

Differential effects of FXR or TGR5 activation in cholangiocarcinoma progression.

Genetic association analysis identifies variants associated with disease progression in primary sclerosing cholangitis.

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