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「"Marzioni M "[Author]」の検索結果

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Current and novel therapeutic opportunities for systemic therapy in biliary cancer.

Cholangiocarcinoma 2020: the next horizon in mechanisms and management.

Impact of reimbursement limits on patient access to direct-acting antivirals in Italy: analysis of data from national registries.

Proteostasis disturbances and endoplasmic reticulum stress contribute to polycystic liver disease: New therapeutic targets.

Soluble CD163 and mannose receptor as markers of liver disease severity and prognosis in patients with primary biliary cholangitis.

Gut-Liver Axis and Inflammasome Activation in Cholangiocyte Pathophysiology.

Patients with Cholangiocarcinoma Present Specific RNA Profiles in Serum and Urine Extracellular Vesicles Mirroring the Tumor Expression: Novel Liquid Biopsy Biomarkers for Disease Diagnosis.

Aging-Related Molecular Pathways in Chronic Cholestatic Conditions.

Aging and the Biological Response to Liver Injury.

Locally acquired hepatitis E virus in Marche Italy: Clinical/laboratory features and outcome.

Secretin/secretin receptor signaling mediates biliary damage and liver fibrosis in early-stage primary biliary cholangitis.

Effects of Vedolizumab in Patients with Primary Sclerosing Cholangitis and Inflammatory Bowel Diseases.

Signaling networks in cholangiocarcinoma: molecular pathogenesis, targeted therapies and drug resistance.

Epidemiology of primary biliary cholangitis in Italy: Evidence from a real-world database.

Ageing-related expression of Twinfilin-1 regulates cholangiocyte biological response to injury.

Wnt-β-catenin signalling in liver development, health and disease.

Inflammation and the Gut-Liver Axis in the Pathophysiology of Cholangiopathies.

Ductular reaction in liver diseases: pathological mechanisms and translational significances.

Pretreatment prediction of response to ursodeoxycholic acid in primary biliary cholangitis: development and validation of the UDCA Response Score.

Knockout of secretin receptor reduces biliary damage and liver fibrosis in Mdr2 mice by diminishing senescence of cholangiocytes.

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