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福岡・大牟田の浸水4mに、防災科研解析…「線状降水帯」相次いで発生 (読売新聞)

防災科学技術研究所(茨城県つくば市)は6日夜、九州北部の大雨で、福岡県大牟田市では浸水の深さが少なくとも4メートルに達したとの解析結果を発表した。 防災科研は、...

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「"Samuelsen JT "[Author]」の検索結果

28件中 1件~20件表示    検索結果をPubMedで見る PubMedで見る

In vitro effects of dental monomer exposure - Dependence on the cell culture model.

Ion release from three different dental alloys - effect of dynamic loading and toxicity of released elements.

Establishing a macrophage model with relevance for oral methacrylate monomer exposures: Attenuated Staphylococcus aureus-induced cytokine release from human macrophages.

The dental monomer HEMA causes proteome changes in human THP-1 monocytes.

The dental monomer 2-hydroxyethyl methacrylate (HEMA) causes transcriptionally regulated adaptation partially initiated by electrophilic stress.

Di-n-butyl phthalate modifies PMA-induced macrophage differentiation of THP-1 monocytes via PPARγ.

Presence and leaching of bisphenol a (BPA) from dental materials.

The dental monomer hydroxyethyl methacrylate (HEMA) counteracts lipopolysaccharide-induced IL-1β release - possible role of glutathione.

Cell toxicity of 2-hydroxyethyl methacrylate (HEMA): the role of oxidative stress.

TEGDMA and filler particles from dental composites additively attenuate LPS-induced cytokine release from the macrophage cell line RAW 264.7.

Cell toxicity of methacrylate monomers-The role of glutathione adduct formation.

Dental monomers inhibit LPS-induced cytokine release from the macrophage cell line RAW264.7.

Mono-2-ethylhexylphthalate (MEHP) induces TNF-α release and macrophage differentiation through different signalling pathways in RAW264.7 cells.

Biotransformation enzymes and lung cell response to 2-hydroxyethyl-methacrylate.

DNA-damage, cell-cycle arrest and apoptosis induced in BEAS-2B cells by 2-hydroxyethyl methacrylate (HEMA).

Role of thiol-complex formation in 2-hydroxyethyl- methacrylate-induced toxicity in vitro.

Role of thiol-complex formation in 2-hydroxyethyl- methacrylate-induced toxicity in vitro.

DNA damage and DNA damage responses in THP-1 monocytes after exposure to spores of either Stachybotrys chartarum or Aspergillus versicolor or to T-2 toxin.

Filler particles used in dental biomaterials induce production and release of inflammatory mediators in vitro.

HEMA reduces cell proliferation and induces apoptosis in vitro.

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