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「"Sommers JA "[Author]」の検索結果

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Mitochondrial genetic variation is enriched in G-quadruplex regions that stall DNA synthesis in vitro.

Peroxide-promoted disassembly-reassembly of Zr-polyoxocations.

Cellular Assays to Study the Functional Importance of Human DNA Repair Helicases.

A high-throughput screen to identify novel small molecule inhibitors of the Werner Syndrome Helicase-Nuclease (WRN).

A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair.

CDK1 phosphorylates WRN at collapsed replication forks.

Biochemical Characterization of the Human Mitochondrial Replicative Twinkle Helicase: Substrate Specificity, DNA Branch-Migration, and Ability to Overcome Blockades to DNA Unwinding.

Biochemical and Cell Biological Assays to Identify and Characterize DNA Helicase Inhibitors.

Catalytic Strand Separation by RECQ1 Is Required for RPA-Mediated Response to Replication Stress.

Close encounters for the first time: Helicase interactions with DNA damage.

Protein Degradation Pathways Regulate the Functions of Helicases in the DNA Damage Response and Maintenance of Genomic Stability.

Impact of Age-Associated Cyclopurine Lesions on DNA Repair Helicases.

DNA Sequences Proximal to Human Mitochondrial DNA Deletion Breakpoints Prevalent in Human Disease Form G-Quadruplexes, a Class of DNA Structures Inefficiently Unwound by the Mitochondrial Replicative Twinkle Helicase.

Novel Function of the Fanconi Anemia Group J or RECQ1 Helicase to Disrupt Protein-DNA Complexes in a Replication Protein A-Stimulated Manner.

Molecular functions and cellular roles of the ChlR1 (DDX11) helicase defective in the rare cohesinopathy Warsaw breakage syndrome.

Targeting an Achilles' heel of cancer with a WRN helicase inhibitor.

Specialization Among Iron-Sulfur Cluster Helicases to Resolve G-Quadruplex DNA Structures that Threaten Genomic Stability.

WERNER SYNDROME HELICASE HAS A CRITICAL ROLE IN DNA DAMAGE RESPONSES IN THE ABSENCE OF A FUNCTIONAL FANCONI ANEMIA PATHWAY.

Human RECQ1 Interacts with Ku70/80 and Modulates DNA End-Joining of Double-Strand Breaks.

Fanconi Anemia Group J Helicase and MRE11 Nuclease Interact to Facilitate the DNA Damage Response.

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