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「"Stravalaci M "[Author]」の検索結果

34件中 1件~20件表示    検索結果をPubMedで見る PubMedで見る

The macrophage tetraspan MS4A4A enhances dectin-1-dependent NK cell-mediated resistance to metastasis.

The Long Pentraxin PTX3 as a Link Between Innate Immunity, Tissue Remodeling, and Cancer.

TNF-stimulated gene-6 (TSG-6) is a key regulator in switching stemness and biological properties of mesenchymal stem cells.

Interaction of C1q With Pentraxin 3 and IgM Revisited: Mutational Studies With Recombinant C1q Variants.

An antipsychotic drug exerts anti-prion effects by altering the localization of the cellular prion protein.

Humanin Specifically Interacts with Amyloid-β Oligomers and Counteracts Their in vivo Toxicity.

Fingolimod Limits Acute Aβ Neurotoxicity and Promotes Synaptic Versus Extrasynaptic NMDA Receptor Functionality in Hippocampal Neurons.

The Anti-Prion Antibody 15B3 Detects Toxic Amyloid-β Oligomers.

Pharmacological inhibition of mannose-binding lectin ameliorates neurobehavioral dysfunction following experimental traumatic brain injury.

A cationic tetrapyrrole inhibits toxic activities of the cellular prion protein.

A New Surface Plasmon Resonance Assay for In Vitro Screening of Mannose-Binding Lectin Inhibitors.

Clusterin binds to Aβ1-42 oligomers with high affinity and interferes with peptide aggregation by inhibiting primary and secondary nucleation.

The new β amyloid-derived peptide Aβ1-6A2V-TAT(D) prevents Aβ oligomer formation and protects transgenic C. elegans from Aβ toxicity.

Scaffold optimization of tetravalent antagonists of the Mannose Binding Lectin.

Exploring the role of MKK7 in excitotoxicity and cerebral ischemia: a novel pharmacological strategy against brain injury.

An acidic microenvironment sets the humoral pattern recognition molecule PTX3 in a tissue repair mode.

A New Surface Plasmon Resonance-Based Immunoassay for Rapid, Reproducible and Sensitive Quantification of Pentraxin-3 in Human Plasma.

Expression of A2V-mutated Aβ in C. elegans results in oligomer formation and toxicity.

Epitope scanning indicates structural differences in brain-derived, monomeric and aggregated mutant prion proteins related to genetic prion diseases.

Novel approaches for studying amyloidogenic peptides/proteins.

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