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「"Szabadkai G "[Author]」の検索結果

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Correction: Impaired cellular bioenergetics caused by GBA1 depletion sensitizes neurons to calcium overload.

Macrophages induce malignant traits in mammary epithelium via IKKε/TBK1 kinases and the serine biosynthesis pathway.

Annual Meeting of the International Society of Cancer Metabolism (ISCaM): Metabolic Adaptations and Targets in Cancer.

Impaired cellular bioenergetics caused by GBA1 depletion sensitizes neurons to calcium overload.

MLH1 deficiency leads to deregulated mitochondrial metabolism.

Fantastic beasts and how to find them-Molecular identification of the mitochondrial ATP-sensitive potassium channel.

Hyperpolarised C MRI: a new horizon for non-invasive diagnosis of aggressive breast cancer.

CHCHD4 regulates tumour proliferation and EMT-related phenotypes, through respiratory chain-mediated metabolism.

Biclustering Analysis of Co-regulation Patterns in Nuclear-Encoded Mitochondrial Genes and Metabolic Pathways.

Metabolic Profiling of Live Cancer Tissues Using NAD(P)H Fluorescence Lifetime Imaging.

Determination of ATP, ADP, and AMP Levels by Reversed-Phase High-Performance Liquid Chromatography in Cultured Cells.

Mitochondrial Permeability Transition: A Molecular Lesion with Multiple Drug Targets.

VHL-Mediated Regulation of CHCHD4 and Mitochondrial Function.

Annual Meeting of the International Society of Cancer Metabolism (ISCaM): Cancer Metabolism.

Mitochondria and Reactive Oxygen Species in Aging and Age-Related Diseases.

Inositol Trisphosphate Receptor Mediated Ca2+ Signalling Stimulates Mitochondrial Function and Gene Expression in Core Myopathy Patients.

Targeting RORs nuclear receptors by novel synthetic steroidal inverse agonists for autoimmune disorders.

NADH Shuttling Couples Cytosolic Reductive Carboxylation of Glutamine with Glycolysis in Cells with Mitochondrial Dysfunction.

Organelles: The Emerging Signalling Chart of Mitochondrial Dynamics.

Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.

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