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「Neurobiol Dis[Journal]」の検索結果

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Apolipoprotein E and oxidative stress in brain with relevance to Alzheimer's disease.

Multielectrode array analysis of EEG biomarkers in a mouse model of Fragile X Syndrome.

The green tea polyphenol epigallocatechin-3-gallate (EGCG) restores CDKL5-dependent synaptic defects in vitro and in vivo.

Dysregulated iron metabolism in C. elegans catp-6/ATP13A2 mutant impairs mitochondrial function.

Using human induced pluripotent stem cells (hiPSCs) to investigate the mechanisms by which Apolipoprotein E (APOE) contributes to Alzheimer's disease (AD) risk.

Influence of Western diet and APOE genotype on Alzheimer's disease risk.

Gene therapy for overexpressing Neuregulin 1 type I in skeletal muscles promotes functional improvement in the SOD1 ALS mice.

Novel presenilin 1 and 2 double knock-out cell line for in vitro validation of PSEN1 and PSEN2 mutations.

Transient upregulation of translational efficiency in prodromal and early symptomatic Tg2576 mice contributes to Aβ pathology.

Alpha-synuclein/synapsin III pathological interplay boosts the motor response to methylphenidate.

UsnRNP trafficking is regulated by stress granules and compromised by mutant ALS proteins.

APOE-amyloid interaction: Therapeutic targets.

Region-specific glial homeostatic signature in prion diseases is replaced by a uniform neuroinflammation signature, common for brain regions and prion strains with different cell tropism.

Corrigendum to "APP-dependent alteration of GSK3β activity impairs neurogenesis in the Ts65Dn mouse model of Down syndrome" [Neurobiology of Disease 67 (2014) 24-36].

The antagonism of prostaglandin FP receptors inhibits the evolution of spreading depolarization in an experimental model of global forebrain ischemia.

N-terminal acetylation mutants affect alpha-synuclein stability, protein levels and neuronal toxicity.

Genetics of Parkinson's disease: An introspection of its journey towards precision medicine.

FMRP regulates presynaptic localization of neuronal voltage gated calcium channels.

Brain insulin resistance triggers early onset Alzheimer disease in Down syndrome.

Neuronally expressed anti-tau scFv prevents tauopathy-induced phenotypes in Drosophila models.

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