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「Nucleic Acids Res[Journal]」の検索結果

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Resolving Toxic DNA repair intermediates in every E. coli replication cycle: critical roles for RecG, Uup and RadD.

3-Dimensional architecture of the human multi-tRNA synthetase complex.

Switching the activity of Taq polymerase using clamp-like triplex aptamer structure.

A critical analysis of methods used to investigate the cellular uptake and subcellular localization of RNA therapeutics.

Variance-adjusted Mahalanobis (VAM): a fast and accurate method for cell-specific gene set scoring.

Bypass of DNA interstrand crosslinks by a Rev1-DNA polymerase ζ complex.

Structures of mammalian GLD-2 proteins reveal molecular basis of their functional diversity in mRNA and microRNA processing.

NC2 complex is a key factor for the activation of catalase-3 transcription by regulating H2A.Z deposition.

Taxonomic classification method for metagenomics based on core protein families with Core-Kaiju.

RADX condenses single-stranded DNA to antagonize RAD51 loading.

Transcription of intragenic CpG islands influences spatiotemporal host gene pre-mRNA processing.

CASC3 promotes transcriptome-wide activation of nonsense-mediated decay by the exon junction complex.

Bulk and single-molecule analysis of a bacterial DNA2-like helicase-nuclease reveals a single-stranded DNA looping motor.

DNA backbone interactions impact the sequence specificity of DNA sulfur-binding domains: revelations from structural analyses.

IMPLICON: an ultra-deep sequencing method to uncover DNA methylation at imprinted regions.

Highly efficient 'hit-and-run' genome editing with unconcentrated lentivectors carrying Vpr.Prot.Cas9 protein produced from RRE-containing transcripts.

Embryonic tissue differentiation is characterized by transitions in cell cycle dynamic-associated core promoter regulation.

Rph1 coordinates transcription of ribosomal protein genes and ribosomal RNAs to control cell growth under nutrient stress conditions.

STing: accurate and ultrafast genomic profiling with exact sequence matches.

A point mutation in the nuclease domain of MLH3 eliminates repeat expansions in a mouse stem cell model of the Fragile X-related disorders.

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