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「Nucleic Acids Res[Journal]」の検索結果

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AtFKBP53: a chimeric histone chaperone with functional nucleoplasmin and PPIase domains.

Phosphorylation of Tet3 by cdk5 is critical for robust activation of BRN2 during neuronal differentiation.

Structural basis of non-canonical transcriptional regulation by the σA-bound iron-sulfur protein WhiB1 in M. tuberculosis.

Same fold, different properties: polarizable molecular dynamics simulations of telomeric and TERRA G-quadruplexes.

Cross-subunit catalysis and a new phenomenon of recessive resurrection in Escherichia coli RNase E.

mRNA regions where 80S ribosomes pause during translation elongation in vivo interact with protein uS19, a component of the decoding site.

FLASH: ultra-fast protocol to identify RNA-protein interactions in cells.

Hybridization-mediated off-target effects of splice-switching antisense oligonucleotides.

The human HELLS chromatin remodelling protein promotes end resection to facilitate homologous recombination and contributes to DSB repair within heterochromatin.

HECTD1 promotes base excision repair in nucleosomes through chromatin remodelling.

Free energy landscape of salt-actuated reconfigurable DNA nanodevices.

Targeted insertional mutagenesis libraries for deep domain insertion profiling.

Ribosomal protein gene RPL9 variants can differentially impair ribosome function and cellular metabolism.

Design of a programmable biosensor-CRISPRi genetic circuits for dynamic and autonomous dual-control of metabolic flux in Bacillus subtilis.

LGP2 virus sensor enhances apoptosis by upregulating apoptosis regulatory genes through TRBP-bound miRNAs during viral infection.

Negative cooperativity between Gemin2 and RNA provides insights into RNA selection and the SMN complex's release in snRNP assembly.

Conformation-dependent restraints for polynucleotides: the sugar moiety.

DSS1 interacts with and stimulates RAD52 to promote the repair of DSBs.

SIRT1/2 orchestrate acquisition of DNA methylation and loss of histone H3 activating marks to prevent premature activation of inflammatory genes in macrophages.

Specific enhancer selection by IRF3, IRF5 and IRF9 is determined by ISRE half-sites, 5' and 3' flanking bases, collaborating transcription factors and the chromatin environment in a combinatorial fashion.

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