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Natriuretic peptide receptor guanylyl cyclase-A pathway counteracts glomerular injury evoked by aldosterone through p38 mitogen-activated protein kinase inhibition.

A Personalized Model of COQ2 Nephropathy Rescued by the Wild-Type COQ2 Allele or Dietary Coenzyme Q10 Supplementation.

Deletion of Inositol-Requiring Enzyme-1α in Podocytes Disrupts Glomerular Capillary Integrity and Autophagy.

Rapamycin Induces Autophagy and Reduces the Apoptosis of Podocytes Under a Stimulated Condition of Immunoglobulin A Nephropathy.

Janus Kinase 2 Regulates Transcription Factor EB Expression and Autophagy Completion in Glomerular Podocytes.

Thrombin-Induced Podocyte Injury Is Protease-Activated Receptor Dependent.

Differential expression profile analysis of PSTK-regulated mRNAs in podocytes.

Urinary podocyte and TGF-β1 mRNA as markers for disease activity and progression in anti-glomerular basement membrane nephritis.

A disease model of diabetic nephropathy in a glomerulus-on-a-chip microdevice.

Cyclosporine A alters expression of renal microRNAs: New insights into calcineurin inhibitor nephrotoxicity.

Molecular mechanisms involved in podocyte EMT and concomitant diabetic kidney diseases: an update.

Metabolism and homeostasis in the kidney: metabolic regulation through insulin signaling in the kidney.

Effect of berberine on the renal tubular epithelial-to-mesenchymal transition by inhibition of the Notch/snail pathway in diabetic nephropathy model KKAy mice.

Activated protein C inhibits neutrophil extracellular trap formation in vitro and activation in vivo.

Novel Roles for Podocalyxin in Regulating Stress Myelopoiesis, Rap1a and Neutrophil Migration.

Fibrinogen links podocyte injury with Toll-like receptor 4 and is associated with disease activity in FSGS patients.

Focal segmental glomerulosclerosis and medullary nephrocalcinosis in children with ADCK4 mutations.

New Insights into Podocyte Biology in Glomerular Health and Disease.

NLRP3 Inflammasome as a Novel Target for Docosahexaenoic Acid Metabolites to Abrogate Glomerular Injury.

Autophagy is activated to protect against podocyte injury in adriamycin-induced nephropathy.

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