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Urinary fatty acid and retinol binding protein-4 predict CKD progression in severe NAFLD patients with hypertension: 4-year study with clinical and experimental approaches.

The anti-oxidative role of cytoglobin in podocytes: implications for a role in chronic kidney disease.

Time-course of sodium transport along the nephron in nephrotic syndrome: The role of potassium.

The Use of High-Throughput Transcriptomics to Identify Pathways with Therapeutic Significance in Podocytes.

Efficacy of a mitochondrion-targeting agent for reducing the level of urinary protein in rats with puromycin aminonucleoside-induced minimal-change nephrotic syndrome.

Targeting ROCK signaling in health, malignant and non-malignant diseases.

Cosmc-dependent mucin-type O-linked glycosylation is essential for podocyte function.

Kidney podocyte-associated gene polymorphisms affect tacrolimus concentration in pediatric patients with refractory nephrotic syndrome.

Detection of Cryptic Mosaicism in X-linked Alport Syndrome Prompts to Reevaluate Living-donor Kidney Transplantation.

Metabolic pathways mediate pathogenesis and offer targets for treatment in rheumatic diseases.

Expression and localization of DAB1 and Reelin during normal human kidney development.

Modification of an aggressive model of Alport Syndrome reveals early differences in disease pathogenesis due to genetic background.

Structure-Function Relationship and Physiological Roles of Transient Receptor Potential Canonical (TRPC) 4 and 5 Channels.

Excessive arachidonic acid induced actin bunching remodeling and podocyte injury via a PKA-c-Abl dependent pathway.

Effects of BSF on Podocyte Apoptosis via Regulating the ROS-Mediated PI3K/AKT Pathway in DN.

Involvement of prohibitin 1 and prohibitin 2 upregulation in cBSA-induced podocyte cytotoxicity.

Phenotypic analysis of mice carrying human-type MAFB p.Leu239Pro mutation.

Curcumin, as a pleiotropic agent, improves doxorubicin-induced nephrotic syndrome in rats.

Platelet thrombus formation in eHUS is prevented by anti-MBL2.

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