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Morphological Features of Minimal Change Disease and Focal Segmental Glomerulosclerosis Using Repeat Biopsy and Parietal Epithelial Cell Marker.

Genetic Ablation of SLK Exacerbates Glomerular Injury in Adriamycin Nephrosis in Mice.

Protective effect of ferulic acid on STZ-induced diabetic nephropathy in rats.

Podocytes: the role of lysosomes in the development of nephrotic syndrome.

EDA2R mediates podocyte injury in high glucose milieu.

Cathepsin C is a novel mediator of podocyte and renal injury induced by hyperglycemia.

Podocyte RNA sequencing reveals Wnt- and ECM-associated genes as central in FSGS.

Lipotoxicity and Diabetic Nephropathy: Novel Mechanistic Insights and Therapeutic Opportunities.

Role of cathepsin L in idiopathic nephrotic syndrome in children.

From protein uptake to Dent disease: an overview of the CLCN5 gene.

Anti-apoptosis mechanism of triptolide based on network pharmacology in focal segmental glomerulosclerosis rats.

Loganin and catalpol exert cooperative ameliorating effects on podocyte apoptosis upon diabetic nephropathy by targeting AGEs-RAGE signaling.

Role of Impaired Nutrient and Oxygen Deprivation Signaling and Deficient Autophagic Flux in Diabetic CKD Development: Implications for Understanding the Effects of Sodium-Glucose Cotransporter 2-Inhibitors.

Circ_0000285 promotes podocyte injury through sponging miR-654-3p and activating MAPK6 in diabetic nephropathy.

Heparanase in Kidney Disease.

Autosomal Dominant Tubulointerstitial Kidney Disease-Uromodulin Misclassified as Focal Segmental Glomerulosclerosis or Hereditary Glomerular Disease.

Triptolide potentiates the cytoskeleton-stabilizing activity of cyclosporine A in glomerular podocytes a GSK3β dependent mechanism.

Rare Occurrence of PLA2R-Positive Membranous Nephropathy in a Patient with Sjogren's Syndrome and CIDP.

Pathophysiologic mechanisms in diabetic kidney disease: A focus on current and future therapeutic targets.

Immunization with Pentraxin3 prevents transition from subclinical to clinical lupus nephritis in lupus-prone mice: Insights from renal ultrastructural findings.

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