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MiR-320a induces diabetic nephropathy via inhibiting MafB.

CD36-Mediated Lipid Accumulation and Activation of NLRP3 Inflammasome Lead to Podocyte Injury in Obesity-Related Glomerulopathy.

Nephrin Signaling Results in Integrin 1 Activation.

The motor protein Myo1c regulates transforming growth factor-β-signaling and fibrosis in podocytes.

Importance of Genetic Diagnostics in Adult-Onset Focal Segmental Glomerulosclerosis.

Intrinsic tumor necrosis factor-α pathway is activated in a subset of patients with focal segmental glomerulosclerosis.

[Molecular mechanisms of insulin resistance and interventional effects of Chinese herbal medicine].

Three-dimensional architecture of pericardial nephrocytes in Drosophila melanogaster revealed by FIB/SEM tomography.

Hydroxychloroquine-induced podocytopathy mimicking Fabry disease.

Ectopic expression of CLDN2 in podocytes is associated with childhood onset nephrotic syndrome.

Magnesium isoglycyrrhizinate ameliorates fructose-induced podocyte apoptosis through downregulation of miR-193a to increase WT1.

Gadolinium-based contrast agents: Stimulators of myeloid-induced renal fibrosis and major metabolic disruptors.

Elimination of the fibrinogen integrin αβ-binding motif improves renal pathology in mice with sickle cell anemia.

Circulating factors cause proteinuria in parabiotic zebrafish.

Disruption of the exocyst induces podocyte loss and dysfunction.

Foxc2 is essential for podocyte function.

Sanqi oral solution ameliorates renal damage and restores podocyte injury in experimental membranous nephropathy via suppression of NFκB.

Disruption of podocyte cytoskeletal biomechanics by dasatinib leads to nephrotoxicity.

Adiponectin for the treatment of diabetic nephropathy.

Molecular classification of membranous nephropathy.

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